alcoholic cardiomyopathy recovery time

The prognosis for individuals diagnosed with ACM largely depends on their commitment to abstaining from alcohol. Studies indicate that individuals who continue to drink after being diagnosed have a significantly poorer outlook. Approximately 40–80% of these individuals may die within 10 years of their diagnosis. Conversely, those who completely quit alcohol generally show improved heart function and overall health Medical News Today.

Complications/side effects of the treatment

Understanding that message is the first step to healing and thriving once again. If the disease is caught early, stopping marijuana addiction alcohol use completely and taking certain medications can help restore the heart’s function. This is especially true if your genetic condition affects how your body metabolizes alcohol. However, the best way to completely prevent alcoholic cardiomyopathy is not to drink at all.

Table 1. List of literature articles reviewed in this study.

alcoholic cardiomyopathy recovery time

They’ll also ensure that you don’t have an underlying issue like cancer or Crohn’s disease that must also be treated. Alcohol consumption is one of several lifestyle factors that can contribute to joint pain. First, continued alcohol consumption can cause peripheral nerve damage, with the alcohol wearing away the protective sheathing of nerve cells. People with sudden yellowed skin (called jaundice) and a history of heavy drinking likely have alcoholic hepatitis. Survival rates used to be significantly lower for late-stage alcoholic liver disease before liver transplants were viable. Alcoholic hepatitis and cirrhosis mean a person has caused extensive damage to their liver as a result of alcohol consumption.

How common is this condition?

alcoholic cardiomyopathy recovery time

When it comes to wine, one drink is defined as a 5-ounce (148 ml) serving, which typically contains about 12% ABV. Distilled spirits, such as vodka, whiskey, rum, or tequila, are measured as 1.5 ounces (44 ml) per drink, with a typical ABV of around 40%. It is important to note that the size and strength of different alcoholic beverages can vary, so these definitions serve as general guidelines. It is always advisable to be mindful of individual tolerance and consume alcohol responsibly 4-6. Alcoholic cardiomyopathy (ACM) is a cardiac disease caused by chronic alcohol consumption.

alcoholic cardiomyopathy recovery time

Over time, chronic alcohol use can lead to more devastating health conditions from osteoporosis and fractures to chronic joint pain and other concerns. However, excessive alcohol consumption can force the cells in a person’s liver to work harder than normal. Over time, this can damage a person’s liver cells, which can cause them to develop health issues.

Alcohol in excessive quantities has a directly toxic effect on heart muscle cells. According to current knowledge, prolonged and excessive alcohol consumption plays a significant role in inducing oxidative stress within the myocardium. This can occur through direct means, by promoting the generation of free radicals, or indirectly, by triggering the release of hormones, such as angiotensin II, or activating other systems. Moreover, alcohol may reduce the levels of transport proteins and diminish antioxidant activity by decreasing the plasma concentration of antioxidant enzymes.

Acetaldehyde damage

Furthermore, there are conflicting data among studies regarding the prognosis of the condition, with some showing overall mortality near 60% and others showing a mortality rate of only 19% (Table 1). Data on the amount of alcohol consumption required to cause ACM are limited and controversial. At present ACM is considered a specific disease both by the European Society of Cardiology alcoholic cardiomyopathy recovery time (ESC) and by the American Heart Association (AHA)18,19. In the ESC consensus document on the classification of cardiomyopathies, ACM is classified among the acquired forms of DCM19.

Prognosis and Outlook

Earlier studies by Puszkin and Rubin (10) were the first to suggest that alcohol had effects on the regulatory proteins, troponins or tropomyosins. They found that high concentrations of alcohol (150 mmol to 180 mmol) administered acutely inhibited calcium binding to troponin-tropomyosin protein complexes in vitro. Also, acute alcohol administration in a rat model significantly raised plasma cardiac troponin T level after 2.5 h (11). In our patient, the elevated troponin T is suggestive of acute myocardial damage. However, the falling level of troponin T in hospital suggested that the myocardial damage had occurred before admission. A case of rapid reversal of alcohol-induced cardiomyopathy with abstinence is reviewed.

alcoholic cardiomyopathy recovery time

Symptoms of Alcoholic Cardiomyopathy

Other studies investigated the catalase levels and activity among rats with ACM with a control group. They demonstrated a much higher catalase activity among rats suffering from ACM. This may be explained by the fact that the increased catalase activity in those who have a long history of alcohol abuse may represent a protective and adoptive reaction to the persistent high ethanol levels 11. A well-functioning heart is essential for maintaining the overall health of virtually every system in the body. Therefore, when the heart’s efficiency is compromised due to alcohol-induced cardiomyopathy, the impacts are pervasive and consequential.

Some studies have suggested that even moderation of alcohol consumption similar outcomes as compared to abstinence. To make a diagnosis, your doctor will perform a physical examination and ask you about your medical history. You should also follow your doctor’s guidance and advice on any treatments you receive. This includes taking your medication as instructed and eating a healthy diet.

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